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CHAMPIONS TACKLING MS - AWARDS Dinner, Honoring Aaron Boster, MD and Jon e. Glaser, DDS - now open for registration. Visit www.events.msvn.org

Thursday, January 12, 2012

MS and 100 Pounds Fade Away Eating Mostly Raw, Vegan Diet


I can somewhat agree with what this person is writing, concerning her veggie diet
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Written by: Sandy Henson Corso


I have been on a serious health journey for more than three years now. Here is my story. This is going to be a shocker because lots of people don't know this entire story. Even some of my family and closest friends don't know what I have been through with regard to my health. I will share the details in hopes of inspiring people to eat more plants and fewer animals. 

In 2000 I was diagnosed with Multiple Sclerosis. It was a very, very dark and scary time. When I tell you I got down lots on my hands and knees and prayed for God's help I am not exaggerating! I was scared, very scared. I walked around like a zombie for years. Then I went on about my life, getting married, buying a house, having two beautiful children. After all this, about 3.5 years ago, I was still really scared. I was not feeling good, I had an IV in my arm receiving intravenous steroids, and I was more than 100 pounds overweight. Plus, every day I had to have my husband give me my medication, which was a shot, for my MS. How could this happen? I was in a dark place.

So I decided to make a change. I went to a personal trainer named Heidi who happened to move right next door where I was working. The universe works in strange ways. I sat with her in tears: How could this be? How could I have allowed myself to be so unhealthy and so overweight? She was amazing and assured me I would be fine. I worked with her for about eight months. I lost some weight and inches but most importantly I made fitness part of my life. My next step was to join a gym and see if I could keep up my fitness. I joined and I did keep up. I started running races. I did 5k races and then 5-mile races. 

I then decided to go off my MS medication. My instincts have always been right for me. I believe 100 percent in the power of my own healing through diet and exercise. I do supplement with vitamin D3 and B12, which I believe are both very important. I am not saying everyone should go off their medication -- you have to do what YOUR instincts tell YOU. 


READ MORE



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Disclaimer:  'MS Views and News' (MSVN), does not endorse any products or services found on this blog. It is up to you to seek advice from your healthcare provider. The intent of this blog is to provide information on various medical conditions, medications, treatments, and procedures for your personal knowledge and to keep you informed of current health-related issues. It is not intended to be complete or exhaustive, nor is it a substitute for the advice of your physician. Should you or your family members have any specific medical problem, seek medical care promptly.
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A First Line, Personalized, Treatment for MS completes phase II studies


information provided to us by Thomas Mann

Tovaxin®

Opexa's lead product candidate is Tovaxin, a personalized T-cell vaccine for the treatment of multiple sclerosis (MS) that is specifically tailored to each patient's disease profile. Opexa believes the potential combination of efficacy, superior safety, improved tolerability and patient-friendly administration schedule may position Tovaxin as the MS treatment of choice as compared to existing therapeutic options.
Tovaxin is designed to reduce the number of specific subsets of autoreactive T-cells known to attack myelin. Tovaxin is manufactured using Opexa Therapeutics’ proprietary method for the production of patient-specific T-cell vaccines, which comprises the collection of blood from the MS patient, the harvest and expansion of disease-causing T-cells from the blood, and the return of these expanded, irradiated T-cells back to the patient. These attenuated T-cells, which comprise the Tovaxin vaccine, are reintroduced into the patient via subcutaneous injection to trigger a therapeutic immune system response.
Tovaxin is being developed as a first line treatment for MS. The treatment will consist of donating blood and creating a vaccine using the patient’s own cells. The vaccine cells will be irradiated to render them unable to divide, but able to evoke an immune response. The vaccines will be administered in the doctor’s office as a subcutaneous injection in the arm given five times a year. The first four injections are administered a month apart with the fifth and final injection being administered two months after the fourth. While many other MS treatments have constant side effects such as flu symptoms, injection site problems, and serious adverse events, Tovaxin has not had any treatment related serious adverse events (SAEs). The most common side effect reported form clinical studies has been mild to moderate injection site reaction that usually resolves without treatment within 24 hours. Tovaxin therapy is specific for the immune response against myelin destroying T cells and does not cause a general immunosuppression. Tovaxin may be a strong alternative for subjects who do not want to start hospital infusions or weekly injections, or have not been able to tolerate the side effects or infections of immunosuppressant drugs on the market. 
Tovaxin could represent a viable alternative for patients who:
  • Have chosen to stop or not start treatments;
  • Are newly diagnosed with MS;
  • Are dissatisfied with currently marketed drugs.

 CONTINUE READING



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Disclaimer:  'MS Views and News' (MSVN), does not endorse any products or services found on this blog. It is up to you to seek advice from your healthcare provider. The intent of this blog is to provide information on various medical conditions, medications, treatments, and procedures for your personal knowledge and to keep you informed of current health-related issues. It is not intended to be complete or exhaustive, nor is it a substitute for the advice of your physician. Should you or your family members have any specific medical problem, seek medical care promptly.
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Understanding and Treating MS Relapses



Article Source: MS Assoc of America


A new MSAA publication, Understanding and Treating MS Relapses, is now available to view, download and print, or order!

Read Understanding and Treating MS Relapses

As with all publications from MSAA, this eight-panel brochure is provided free of charge to the MS community and to anyone interested in learning more about multiple sclerosis (MS).
MSAA's latest publication gives a concise but thorough explanation of:
  • How to recognize a relapse
  • How to differentiate it from a "pseudoexacerbation"
  • When to see a neurologist
  • Treatment options
In the latter section, FDA-approved treatments as well as experimental treatments are described. A brief section at the end provides important tips on treatment monitoring and follow-up, both with your doctor and at home.
We hope that you will take advantage of this new, free publication! Please read, download, or print a copy of Understanding and Treating MS Relapses today! You may also request a free copy to be mailed to your home or call MSAA at (800) 532-7667 for more information or to order a copy.


Please download or order our new publication today! And if you find that you have additional questions about MS, please email MSquestions@msassociation.org.

If links are disabled in this email:

To read, download, or print Understanding and Treating MS Relapses, copy and paste the following into your web browser: support.msassociation.org/relapses




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Disclaimer:  'MS Views and News' (MSVN), does not endorse any products or services found on this blog. It is up to you to seek advice from your healthcare provider. The intent of this blog is to provide information on various medical conditions, medications, treatments, and procedures for your personal knowledge and to keep you informed of current health-related issues. It is not intended to be complete or exhaustive, nor is it a substitute for the advice of your physician. Should you or your family members have any specific medical problem, seek medical care promptly.
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Saskatchewan sends MS patients to U.S. for clinical trial of liberation therapy



REGINA - Multiple sclerosis patients in Saskatchewan can apply to take part in a clinical trial of a controversial treatment that has been touted as a potential therapy.
Premier Brad Wall said a New York state doctor is in the final stages of getting U.S. federal approval for what's believed to be the largest liberation therapy trial of its type.
The province is providing $2.2 million so 86 MS patients can be included in the two-year, double-blind trial at the Albany Medical Center, said Wall.
The procedure is not currently offered in Canada and some patients have travelled around the world to seek it out despite it having not been proven to work.
"While we're talking here, people are raising money and having bake sales and getting the support of neighbours and friends to try to go somewhere to have the treatment. Well, we should find some answers," said Wall.
The double-blind aspect of the study means only half of the patients will actually receive the treatment. The patient and the physicians who do the followup will not know if they got the treatment.
"They'll knowingly volunteer for the trials understanding that they may not get the treatment, which of course is probably what many would like to have. But they're going to do it in the interest of science, in the interest of demonstrating the efficacy of this treatment or coming to some other conclusion, which would also be helpful," said Wall.
Applicants have until Feb. 24 to apply either through the government's website or a toll-free line.


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Disclaimer:  'MS Views and News' (MSVN), does not endorse any products or services found on this blog. It is up to you to seek advice from your healthcare provider. The intent of this blog is to provide information on various medical conditions, medications, treatments, and procedures for your personal knowledge and to keep you informed of current health-related issues. It is not intended to be complete or exhaustive, nor is it a substitute for the advice of your physician. Should you or your family members have any specific medical problem, seek medical care promptly.
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Wednesday, January 11, 2012

Study Shows Potential of Lab Test to Detect Virus Which Causes PML in People with MS – ongoing study may help identify risk for PML in people treated with natalizumab



Biogen Idec researchers have published results on a blood test that detects antibodies to the JC virus, the virus responsible for PML (a severe brain infection). PML has emerged in some people who have taken natalizumab (Tysabri,® Biogen Idec and Elan; Read more about natalizumab and PML). This paper (Annals of Neurology 2011;70:742-750) reports that in an ongoing study of 1,096 people in the U.S. with relapsing MS being treated or considering treatment with Tysabri, 56% had evidence of JC virus antibodies. The presence of antibodies indicates that a person has at some point been infected by or exposed to the virus, which usually lies dormant. Ultimately the study may show whether detection of antibodies to JC virus can predict an individual’s risk for developing PML and help guide treatment decisions.
Details: In studies of patients who have developed PML on Tysabri, all of those tested had serum antibodies prior to the onset of PML. The company has been conducting ongoing studies of its two-step laboratory test (called STRATIFY JC virus™) to determine whether the incidence of PML in Tysabri-treated patients is lower in those who do not have detectable antibodies to JC virus.
In this study, called STRATIFY-1, women had a lower prevalence of antibodies than men (53.4% versus 64.3%), and antibodies were more prevalent with older age. The false-negative rate was 2.7%, meaning that in about 3 out of 100 times, the lab test failed to detect JC virus antibodies in patients who had evidence of the virus DNA detected by a urine test.
The company is continuing to test people starting or already taking natalizumab therapy to determine whether the lab test can reliably predict a person’s risk for developing PML, which may help doctors and patients make more informed treatment choices. Read more about the STRATIFY study.

article source: NMSS


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Disclaimer:  'MS Views and News' (MSVN), does not endorse any products or services found on this blog. It is up to you to seek advice from your healthcare provider. The intent of this blog is to provide information on various medical conditions, medications, treatments, and procedures for your personal knowledge and to keep you informed of current health-related issues. It is not intended to be complete or exhaustive, nor is it a substitute for the advice of your physician. Should you or your family members have any specific medical problem, seek medical care promptly.
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Tuesday, January 10, 2012

Spasticity gene finding provides clues to causes of nerve cell degeneration


January 9, 2012
The discovery of a gene that causes a form of hereditary spastic paraplegia (HSP) may provide scientists with an important insight into what causes axons, the stems of our nerve cells, to degenerate in conditions such as multiple sclerosis.
n the  today, an international team of scientists led by Dr Evan Reid at the University of Cambridge, and Dr Stephan Zuchner from the University of Miami, report that mutations in the gene known as 'reticulon 2' on chromosome 19 cause a form of HSP, a condition characterised by progressive  and  (spasticity) of the legs, caused by selective and specific degeneration of axons
The team identified three mutations in the reticulon 2 gene as causing a type of HSP – in one case, this mutation included an entire deletion of the gene. In addition, the researchers showed that reticulon 2 interacts with another gene, spastin.  in this latter gene cause the most common form of hereditary spastic paraplegia.
Reticulon 2 provides the genetic code for a reticulon protein that is a member of a family of proteins recently shown to play a key role in shaping the endoplasmic reticulum. The endoplasmic reticulum is a network of interconnected sheets and tubules that extends throughout the cytoplasm in nearly all cells. It has a number of functions, including protein synthesis, calcium signalling and regulation of other components of the cell. Recent data suggest that the sheets are involved in protein synthesis, whereas the tubules are specialised to carry out the other functions.
This new study provides the most direct evidence to date that defects in how the endoplasmic reticulum is shaped and formed could underlie axon degeneration. When axons degenerate, signals are unable to pass through the , leading to a breakdown of communication within the central nervous system. This is common in degenerative diseases of the nervous system, such as multiple sclerosis.
"Our work highlights important new disease mechanisms, which may provide a platform for us to study how axons are damaged in devastating illnesses such as HSP, and perhaps even in , which in some cases is very similar to HSP," explains Dr Reid, a Wellcome Trust Senior Research Fellow in Clinical Science. "But we must not forget how this work may immediately directly benefit families affected by hereditary spastic paraplegia, for whom the discovery now opens up the possibility of genetic counselling and testing."
Article source: Medical Press
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Disclaimer:  'MS Views and News' (MSVN), does not endorse any products or services found on this blog. It is up to you to seek advice from your healthcare provider. The intent of this blog is to provide information on various medical conditions, medications, treatments, and procedures for your personal knowledge and to keep you informed of current health-related issues. It is not intended to be complete or exhaustive, nor is it a substitute for the advice of your physician. Should you or your family members have any specific medical problem, seek medical care promptly.
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Common virus may trigger MS nerve damage



FOR the first time we have an answer to what Epstein-Barr virus (EBV) may be doing in the brains of people with multiple sclerosis. RNA from the virus could be triggering the inflammation of nerve tissue characteristic of the disease.
EBV is present in almost all people with MS, and having glandular fever, caused by the virus, is known to increase the risk of developing MS.
To investigate further, Ute-Christiane Meier at Queen Mary University of London and her colleagues performed a post-mortem analysis of the brains of people with and without MS. They detected EBV in damaged areas of the MS brains. Antibody tests showed that the virus hadn't been actively spreading, but it had released small RNA molecules into nearby areas. Further studies showed this RNA could activate the immune system, triggering inflammation, which could damage nerve cells and cause the symptoms of MS (NeurologyDOI: 10.1212/wnl.0b013e31823ed057).
"If we can pinpoint EBV as a trigger, it's possible that we could potentially prevent the condition by treating the virus," says Meier.

article source: New Scientist


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