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Saturday, July 15, 2017

First study shows tie between probiotic and improved symptoms of depression


                                                                  
  
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Probiotics may relieve symptoms of depression, as well as help gastrointestinal upset, research from McMaster University has found.



In a study published in the medical journal Gastroenterology (May 2), researchers of the Farncombe Family Digestive Health Research Institute found that twice as many adults with  (IBS) reported improvements from co-existing depression when they took a specific probiotic than adults with IBS who took a placebo.
The study provides further evidence of the microbiota environment in the intestines being in direct communication with the brain said senior author Dr. Premysl Bercik, an associate professor of medicine at McMaster and a gastroenterologist for Hamilton Health Sciences.
"This study shows that consumption of a specific probiotic can improve both gut symptoms and psychological issues in IBS. This opens new avenues not only for the treatment of patients with functional bowel disorders but also for patients with primary psychiatric diseases," he said.
IBS is the most common gastrointestinal disorder in the world, and is highly prevalent in Canada. It affects the large intestine and patients suffer from abdominal pain and altered bowel habits like diarrhea and constipation. They are also frequently affected by chronic anxiety or depression.
The pilot study involved 44 adults with IBS and mild to moderate anxiety or depression. They were followed for 10 weeks, as half took a daily dose of the probiotic Bifidobacterium longum NCC3001, while the others had a placebo.
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Multiple Sclerosis: Making Connections and Shared Decision Making


                                                                  
  
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A dialogue that you will be quite comfortable listening-to, while learning more of the topics being presented:
 


          MS Views and News is MAKING an IMPACT for those, affected by Multiple Sclerosis
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Traveling with Multiple Sclerosis (MS)


                                                                  
  
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A diagnosis of MS shouldn't keep you from participating in activities you enjoy, or even traveling. The key is research and planning. And know your options in terms “mobility” and “accessibility” to get where you want to go and do what you want to do — comfortably, safely, and with a limited drain on your energy.

Recreation

When you're ready to hit the road — or the high seas — get in the know first. Some thoughts:

Travel

Our travel resources publication (.pdf) and the links below an help you started planning your next trip or activity:

Share your air travel experiences

2016 is the 30th anniversary of the Air Carrier Access Act, a federal law that prohibits commercial airlines from discriminating against passengers with disabilities, and requires certain accommodations. The Society — along with other advocacy organizations — is pursuing ways to improve the air travel experience for people with disabilities.
Share your air travel experiences at airaccess30.org.

Air travel regulations are subject to change. Visit tsa.gov/accessibility for updates.

Accessibility regulations

The 2010 amended American's with Disabilities Act (ADA) regulations include new or expanded provisions for nondiscrimination policies. They cover:
  • the use of wheelchair and other power-driven mobility devices
  • the use of service animals
  • reserving and guaranteeing accessible hotel rooms
  • selling tickets for wheelchair accessible seating at sporting events and performance venues
  • guidelines on how the new regulations affect existing facilities
Read more about the ADA and people with MS (.pdf).



MS Views and News is MAKING an IMPACT for those, affected by Multiple Sclerosis
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Thursday, July 13, 2017

MS has changed video - by Aaron Boster, MD


                                                                  
  
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Boster narrates discussion about how everything in MS had changed...except the way in which we see patients: 



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Promo:
Multiple Sclerosis patients and caregivers are invited to join us on December 2, 2017 to listen & learn from Donald Negroski; MD, Megan R. Weigel, DNP, ARNP-c, MSCN; and Aaron Boster, MD; as they provide essential MS information at our upcoming: "2017 Neurological Aspects of MS Symposium" 

For information or to RSVP, click here - thank you


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Multiple sclerosis and fatigue: A review on the contribution of inflammation and immune-mediated neurodegeneration


                                                                  
  
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Review article
Patejdl R, et al. Autoimmun Rev. 2016.

Abstract

Multiple sclerosis (MS) is an immune mediated disease of the central nervous system (CNS) and the leading cause of non-traumatic disability among young and middle-aged adults in the western world. One of its most prevalent and debilitating symptoms is fatigue. Despite the general acceptance of the idea of an immune pathogenesis of MS itself, the role of autoimmunity in the course of MS-fatigue is a matter of debate. Both immune-related processes (acute inflammation, chronic inflammation, immune-mediated neurodegeneration, immune-mediated alterations of endocrine functions related to fatigue) and presumably non-immune-mediated disturbances and factors (sleep disturbances, depression, cognitive alterations, chronic infections, adverse effects of medications) contribute to the clinical picture. 
Data from in vitro and animal experiments has provided evidence for a role of cytokines as IL-1 and TNF-alpha. This association could not be verified directly in blood samples from humans whereas whole blood stimulation protocols gave some indirect evidence for a role of cytokines in MS-fatigue. MRI being able to detect acute and chronic immune mediated damage to the CNS could depict that global atrophy of gray or white matter does not correlate with fatigue. Rather, distinctive clusters of lesions and atrophy at different locations, mostly bifrontal or in subcortical structures, correlate specifically with fatigue. Regardless of the difficulties in pinpointing the immunogenesis of MS-fatigue, an important role of autoimmunity is strongly supported by an indirect route: 
A growing amount of data shows that the highly effective immunotherapeutics which have been introduced to MS-treatment over the last years effectively and sustainably stabilize and ameliorate fatigue in parallel to their dampening effects on the neuroinflammatory process. This review summarizes the existing data on the relation between inflammation, patterns of CNS-lesions and the effects of immunotherapeutics on MS-fatigue.
Copyright © 2015. Published by Elsevier B.V.

PMID

 26589194 [PubMed - indexed for MEDLINE]

Full text






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Spasticity in multiple sclerosis: Contribution of inflammation, autoimmune mediated neuronal damage and therapeutic interventions.


                                                                  
  
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Review article
Patejdl R, et al. Autoimmun Rev. 2017.

Abstract

In contrast to other diseases that go along with spasticity (e.g. spinal cord injury), spasticity in chronic autoimmune diseases involving the CNS is complicated by the ongoing damage of neuronal networks that leads to permanent changes in the clinical picture of spasticity. Multiple sclerosis (MS) is the most frequent autoimmune disease of the central nervous system (CNS) and spasticity is one of the most disabling symptoms. It occurs in more than 80% MS patients at some point of the disease and is associated with impaired ambulation, pain and the development of contractures. 
Besides causing cumulative structural damage, neuroinflammation occurring in MS leads to dynamic changes in motor circuit function and muscle tone that are caused by cytokines, prostaglandins, reactive oxygen species and stress hormones that affect neuronal circuits and thereby spasticity. 
The situation is complicated further by the fact that therapeutics used for the immunotherapy of MS may worsen spasticity and drugs used for the symptomatic treatment of spasticity have been shown to have the potential to alter immune cell function and CNS autoimmunity itself. This review summarizes the current knowledge on the immunologic pathways that are involved in the development, maintenance, dynamic changes and pharmacological modulation of spasticity in MS.
Copyright © 2017. Published by Elsevier B.V.

PMID

 28698092 [PubMed - as supplied by publisher]



Full text


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          MS Views and News is MAKING an IMPACT for those, affected by Multiple Sclerosis
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Sunday, July 9, 2017

How "OCREVUS Access Solutions" Helps the MS Patient


                                                                  
  
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OCREVUS Access Solutions

Your resource for effective access and reimbursement services for OCREVUS™ (ocrelizumab)    - CLICK HERE to access this Patient website

We work in 3 ways to connect you to the medicine you need.


Checking your coverage and costs

We can find out if your health insurance plan covers your Genentech medicine and how much your out-of-pocket costs will be.


Helping you pay for your medicine

We can refer you to financial assistance options to help you pay for the medicine you need.


Working to get your medicine to you

We work with your doctor’s office and/or your specialty pharmacy to get your medicine to you.


LEARN MORE - CLICK HERE



MS Views and News - Provides beneficial information to the Multiple Sclerosis community